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Using postmortem inferior temporal brain cortex tissue from deceased subjects from the Rush Memory and Aging Project (MAP) ( N = 608), we found that the association of iron with pathologically confirmed clinical Alzheimers disease was stronger among those with the adverse APOE -4 allele

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Interestingly, knockdown of Fasn , Scd1 , or the combination of both together with Srebf1 significantly decreased lipid accumulation in primary hepatocytes to a degree comparable to that observed upon GSH depletion (Figure 5N)